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Description
transglycation properties of carnosine, allowing it to compete for the glycating agent, protecting proteins (lens crystallins) against modification

( 200 500 )

5, 75 Thus, data in hypertrophied hearts with normal systolic function do not support a model that impaired ATP-producing capacity might be an antecedent to the transition to decompensated HF

Nat Commun 11 , 5959 (2020)
