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The regulatory mechanisms of ferroptosis are not isolated but are intimately linked to the core metabolic activities of cells

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These effects were accompanied by enhanced hepatic fatty acid catabolism and a reduction in the tissue n-6/n-3 PUFA ratio, underscoring the role of n-3 PUFAs in counteracting chronic TNF-driven inflammation and metabolic dysfunction ( Beyond general inflammation, EPA has also demonstrated a targeted ability to inhibit specific pro-tumorigenic pathways