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This semi-folded structural state enhances the binding of apoE4 to larger lipid-containing particles in plasma and A deposits in the brain (Chetty et al., 2019), predisposing the aggregation of interacting proteins, e.g., seeding of A fibrils

FIGURE 3 Membrane Perturbing Effects of LCACs LCAC membrane incorporation has been modeled in purified phospholipid vesicles, with findings suggesting that LCACs alter the biophysical properties of cardiomyocyte membranes in a manner that could be linked to electrophysiological derangements (Fink and Gross, 1984

Induction of apoptosis by epigallocatechin gallate and autophagy inhibitors in a mouse macrophage-like cell line

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