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glutathione ros mechanism depletion induces oxidative injury and apoptosis via TRPM2 channel activation in renal collecting duct cells Identification of cytotoxic, glutathione-reactive moieties

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doi: 10.1126/science.abn9257

glutathione ros mechanism depletion induces oxidative injury and apoptosis via TRPM2 channel activation in renal collecting duct cells Identification of cytotoxic, glutathione-reactive moieties

M., Mullally, J

glutathione ros mechanism depletion induces oxidative injury and apoptosis via TRPM2 channel activation in renal collecting duct cells Identification of cytotoxic, glutathione-reactive moieties

Alteration of striatal glutamate release after glutamine synthetase inhibition

glutathione ros mechanism depletion induces oxidative injury and apoptosis via TRPM2 channel activation in renal collecting duct cells Identification of cytotoxic, glutathione-reactive moieties

Bone Marrow Transplant 4 Suppl 1:86-9, 1989

glutathione ros mechanism depletion induces oxidative injury and apoptosis via TRPM2 channel activation in renal collecting duct cells Identification of cytotoxic, glutathione-reactive moieties

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