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It is a well-established model for osteoarthritis, especially in cohorts of older age than we used here (52w) (Botter et al., 2011) and for the unhealthy loss in muscle (sarcopenia) and fat mass(Wijnhoven et al., 2005)
Improved animal models are needed to mimic human pathophysiology and forecast results accurately
In this context, DELE1 is not cleaved by OMA1, but rather is stabilized on mitochondria due to mitochondrial import arrest 157
& Chandy, A
