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Targets specific areas of damage including muscles, tendons, ligaments, and the gut
[ 44 ]) utilizing EGFRs
Overall, the application of these neurons successfully models key pathological attributes that are characteristic of AD, including aggregation of amyloid beta, modified production of A40 or A42, hyperphosphorylation of TAU, reduction in synapses and their plasticity as well as amplification of oxidative stress [44]
