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A prior study identified six putative phosphorylation sites (S155, T189, T216, S246, S284, T292) within ArfGAP1 by mass spectrometry yet no quantitative analysis was provided, and it was unclear which LRRK2 variants were used in these assays, including whether comparison to a kinase-inactive LRRK2 negative control was included (Xiong et al., 2012)

DaRezziS.et al (2014)

Combined delivery of CD40 co-stimulation inhibitors using TRAF6i-HDL nanoparticles further disrupts both trained immunity and co-stimulatory pathways in myeloid cells, significantly improving transplant acceptance and inducing long-term immune tolerance (176)

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