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On the other hand, the T-to-C substitution at GCLC rs1555903 leads to higher GCLC gene expression, as shown in a small set of kidney cortex samples (N = 73, = 0.73, P = 0.008) from the Genotype-Tissue Expression (GTEx) dataset 30

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SeMet can also be metabolised via the multi-step trans-sulfuration pathway to selenocysteine, in turn degraded to hydrogen selenide for subsequent methylation by methyltransferases that give rise to methylselenol (Fig