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This imbalance triggers a chain of pathophysiological events that lead to enzymatic dysfunction in endothelial homeostasis, inflammation, and cell proliferation
doi: 10.3390/ijms19103125
57 In Vero cells (ATCC CCL-81) infected with porcine H1N1 strain, N-acetylcysteine was reported to exert a dose-dependent inhibition on virus replication
(2014) indicated that NaHS treatment reduced the release of inflammatory cytokines by inhibiting NF-B activity in the kidney tissues of DN rats, suggesting that H 2 S can alleviate inflammatory response in DN
