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Body skin is generally thicker and less sensitive than facial skin, potentially tolerating higher concentrations
Omaveloxolone functions by inhibiting the Keap1-mediated degradation of Nrf2, thereby facilitating its nuclear translocation and the subsequent transcription of ARE-dependent genes
Taken together, these results confirm the involvement of CD38 in cancer cell proliferation, chemotaxis and survival, and highlight its potential as a target for therapy in CLL [33]
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