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The literature data on atherosclerosis highlight that native CRP and native LDL interact with each other only if either one is immobilized, modified, or aggregated, thus raising the possibility that CRP and LDL can interact with each other under certain pathological conditions, while several studies suggest that pCRP binds to oxidized LDL through its phosphocholine moiety or via its amyloid-like structures, and sometimes when it is complexed with 2 glycoprotein I [120]

Ueland, 2011), and risk of incident CVD was not be supported in meta-analysis, and plasma choline/betaine levels predicted risk of future MACE only when TMAO was elevated (Wang et al., 2014