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Description
Deficient glutamate clearance, usually associated with downregulation of EAAT transporters is one of the primary mechanisms of glutamate excitotoxicity leading to neuronal death (see Table 2 and the previous section)
Ruedemann 1962), which proves abnormalities in the process of photoreceptor hyperpolarization, as well as depolarization disorders of Muller glial cells with disturbances in transmission between photoreceptors, bipolar cells, and RGCs, respectively (Eells et al
The depth of chemical time and the power of enzymes as catalysts[J]
