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Clockophagy is recently discovered as a type of autophagic degradation of aryl hydrocarbon receptor nuclear translocator-like (ARNTL) during ferroptosis, induced by type 2 ferroptosis inducers (e.g., RSL3 and FIN56) [22, 63]

This means contents, including archaea, are not properly moving through and out of the small intestine, potentially causing an increase in the number of bacteria or archaea (i.e

Nicotinamide mononucleotide (NMN)

The activation of the IL-17 pathway, which leads to inflammation and plays a significant role in cholestasis potentially induced by gallstone disease, was also observed (Tedesco et al., 2018)