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Acknowledgment The graphical abstract and figures were partly generated using Servier Medical Art, provided by Servier, licensed under a Creative Commons Attribution 3.0 unported license
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therefore, LCR is essential for mitochondrial -oxidation 34
TNF- has been shown, mainly in experimental ED models and chronic inflammatory or metabolic conditions, to activate caspase-dependent apoptotic pathways while inhibiting the PI3K/Akt survival pathway (134)
