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Without sufficiently bioavailable iron, mitochondrial respiration is impaired, leading to reduced ATP synthesis, which is required to fuel glutathione (GSH) synthesis, a key co-factor required for ferroptosis defence [29,44]

At the center of this regulation sits the endothelium, the thin layer of cells lining every blood vessel in your body

This layer is formed by high molecular weight glycoproteins called mucins (MUC) that are synthesized and secreted by goblet cells

Two more interesting candidates that were more highly expressed in seal than in mouse neurons, although with less margin, are heme oxygenase 2 (HMOX2, FC = 2.5) and paraoxonase 2 (PON2, FC = 4, Supplementary Table S2)