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As a retrograde signaling molecules, mitochondrial-released ROS induce the activation of intracellular redox-related signaling pathways, such as those involving NRF2/Keap1, forkhead box O proteins (FoxOs), heat shock factors, or antioxidant enzymes such as peroxiredoxin 2, promoting an adaptive response aimed at protecting the cells from further damage (Ristow, 2014
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