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Thus, N800 resulted in mouse mortality mainly due to acute liver, kidney, and spleen damages

The lack of an increase in GSH levels in people with normal baseline GSH, despite NAC treatment and an abundance of cysteine, could be explained as occurring through a negative feedback mechanism that regulates the GSH biosynthesis pathway [64]

Hawley 2015)

[DOI] [PubMed] [Google Scholar] 38.Miseta A., Csutora P
