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Description
This can be achieved by reducing intracellular ROS production through low levels of mitochondrial respiration, elevated dependency on glycolytic phenotype, or enhanced capacity of antioxidant detoxification [58, 59]
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On the mechanism of inhibition of DNA-cytosine methyltransferases by cytosine analogs
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The International Journal of Neuropsychopharmacology
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[DOI] [PMC free article] [PubMed] [Google Scholar] 27.Katsube M, Ishimoto T, Fukushima Y, et al
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