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The dependency on KRAS signaling varies across different KRAS mutant cancer types and could reflect the variability in the tumor response, representing a possible mechanism of intrinsic resistance (75, 99)
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These designs allow researchers and manufacturers to target specific biological pathways or interactions
However especially in adult-onset patients, a preserved mucosal architecture characterized by dense lymphocytic infiltration, with no villous atrophy or crypt hyperplasia can also be observed
