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Consequently, the inactivation of GPX4, either through the inhibition of System Xc or the depletion of intracellular GSH, represents a classic experimental method for inducing ferroptosis

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developed a model of Ox-LDL-induced ferroptosis in HUVECs, showing that CTRP13 intervention increased antioxidant enzyme expression, preventing AS and protecting endothelial cells from ferroptosis ( / mice by reducing mitochondrial damage, boosting glutathione and superoxide dismutase levels, and decreasing LPO and MDA levels