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After a 48 h exposure to 100 M H 2 O 2 in serum-free medium, TM3 Leydig cells showed an increased senescence-associated -galactosidase (SA--gal) activity (Figure 3A)
NADPH contributes to recycling in oxidized antioxidant systems involving glutathione, thioredoxins, glutaredoxins, and peroxiredoxins and maintains another reactive oxygen species (ROS)-detoxifying enzyme, catalase, in its active form, suggesting that the homeostatic level of G6PD-derived NADPH determines the rate of oxidative damage [8]
N., Jair, K
Silla, A
