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2011;16(5):e635-e640
Strutz F, Zeisberg M, Renziehausen A, Raschke B, Becker V, van Kooten C, et al
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At the molecular level, ginsenosides mediate these effects primarily by activating the AMPK/SIRT1/PGC1- axis to promote mitochondrial biogenesis, engaging the PI3K/Akt/mTOR pathway to support muscle protein anabolism, and suppressing NF-B-driven inflammatory signaling to limit exercise-induced muscle damage
