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Description
This model proposes that mGSH depletion due to its covalent binding to NAPQI acts as a first-hit, which is followed by a burst of mitochondrial ROS production that activates a second hit involving mitogen-activated protein kinases (MAPKs) that in turn amplifies the wave of mitochondrial ROS generation (Lemasters, 1998)
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Athymic nude mice purchased from GemPharmatech (Nanjing, China)
doi: 10.1186/s12951-024-02314-w 255 LuoJWangXShiZZengYHeLCaoJet al
