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Chikusetsu saponin IVa attenuates isoprenaline-induced myocardial fibrosis in mice through activation autophagy mediated by AMPK/mTOR/ULK1 signaling
181 These findings suggest that mTORC1 can effectively perceive glucose- and energy-related challenges through multiple molecular pathways
Mitochondrial dysfunction is also linked with a metabolic shift from OXPHOS to glycolysis in inflammatory immune cells (74, 75)
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