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Additionally, treatment of HCT116 cells with 5-FU and oxaliplatin slightly decreased p-AKT and p-p38 pathways in HCT116 cells while being prone to increasing p-AKT and p-p38 in HCT116 GSTA4 cells
In hypoxia, the increased expression of the mitochondrial gene might involve the phosphorylation of the AKT signaling pathway, rather than the activation of ERK that is activated in a traumatic neuronal injury through PKC (77)
10.1016/j.resp.2014.07.011 Respir
