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In the latter process, stress-induced activation of the GC-inducible molecule, TSC22D3 [glucocorticoid-induced leucine zipper (GILZ)] in tumour-infiltrating DCs, which inhibited type I interferon (IFN) responses in those cells and IFN- + T-cell activation, was suggested as a possible mechanism [24, 183]
We also discuss recent and ongoing clinical studies investigating the effects of choline supplementation and the potential use of choline-derived metabolites as biomarkers for therapy response
doi: 10.18632/oncotarget.23180, 203
