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We hypothesize that optimal sleep duration (OSD) is reflected in sleep time when sleep homeostasis resolves sleep debt through the compensatory mechanism of extended sleep

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For example, it is possible that older or more depression-prone individuals systematically report worse subjective ratings of sleep even in the absence of objective alterations

Experiments in rat models indicated a potential reduction in reactive oxygen species buildup and associated cell damage, while cell-based studies noted improved neuronal survival under excitotoxic stressors