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Disruption of lung iron homeostasis appears to be involved in several lung diseases and we therefore looked for evidence of ferritinophagy, which describes a form of selective autophagy characterised by degradation of intracellular ferritin mediated by nuclear receptor coactivator 4 (NCOA4), which increases intracellular iron levels leading to accumulation of lipid ROS and ultimately cell death [52]

[DOI] [PubMed] [Google Scholar] 68.Handique J.G., Baruah J.B

One such potential way the acetylation of CoA could alter gene expression would be through chemical recycling of the acetyl-CoA in the cell (Figure 3B)