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Studies showed that the lethality of low levels of CIL56 could be effectively prevented by antioxidants and iron chelators
For research applications Research use may require different concentrations based on experimental protocols
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Xue et al., 2017) and attenuates high glucose-induced central nervous system injury in vitro through the activation of Nrf2 signaling, displaying protective effects against mitochondrial dysfunction-derived OS (Yang et al., 2017)

Structural homology modeling indicated a conserved structure among Egp38, EmMPK2, and H
