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Description
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Klouda, T., Raybagkar, D., Bernstein, B
One study showed, for example, that fibroblasts from ARDS patients are more prone to ferroptosis than normal fibroblasts themselves (58)
To cope with the elevated oxidative stress they generate, cancer cells enhance their antioxidant capacity, largely through sustained activation of the transcription factor NRF2 often driven by mutations in its negative regulator KEAP1
