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Alternatively, prodrugs activated by other processes specific to cancer cells can be used for the depletion of GSH
Dalton, T
120 Using a mouse ischemia model, a previous study has demonstrated that -catenin-mediated signaling was activated in cardiac macrophages, especially in pro-inflammatory subsets
This is the first comprehensive report in PD demonstrating significant GSH depletion as well as concomitant iron enhancement in the SN region
