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Recently, daily intranasal NBP treatment provided protective and neurogenic/angiogenic effects in the post-stroke brain accompanied by functional improvements after a focal ischemic stroke in mice [164]
The Ki in the CMP and ATP kinetic analysis of CMPK was 0.37 M and 0.06 M, respectively (Table 1)
This structural destabilization activates the CREB3ARF4mediated Golgi stress response, which upregulates pro-inflammatory and pro-apoptotic genes including CHOP, thereby exacerbating cellular injury

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