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Gut 67, 12691279

that AFB1, as the parent compound, inhibited rat liver mitochondrial electron transport at the antimycin A-sensitive site (10) and postulated subsequently (5) that the enhanced liver cell necrosis and acute toxicity observed in the low-protein animals could be due to the low rate of metabolism and clearance of AFB1 with a consequent effect on mitochondrial respiration

Volpato A, Da Silva AS, Crecencio RB, Tomasi T, Fortuoso BF, Ribeiro MP, et al

Since a succinyl-group transfer introduces a relatively large structural moiety (100 kDa) compared to lysine methylation and acetylation [16], it has been proposed that succinylation modification could induce a relatively large shift in the charge and the structural alteration with significant effects on the function of the target protein