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These results indicate that while mTORC1 signaling is important for Th1 differentiation, the downstream target of mTOR, Hif-1, may play a role in limiting effector function in these cells
doi: 10.3390/ijerph17082647
Notably, our findings reveal a sophisticated adaptation mechanism wherein the bacterium recalibrated its metabolic pathways in response to GR deficiency without fully restoring virulence
Characterization of portal lymphocytic infiltrates in feline liver
