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The presence of such isoforms likely compensates the absence or low number of Ser-GSTs in these organisms (1 isoform for both C
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Immobilization methods can be categorized as follow (Figure 11) [7]
Differential effects of cigarette smoke on oxidative stress and proinflammatory cytokine release in primary human airway epithelial cells and in a variety of transformed alveolar epithelial cells
