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Hence, GSH depletion is not a relevant signaling mechanism after exposure to low, subtoxic ITC doses while treatment with cytotoxic ITC concentrations potentially triggers low GSH responsive genes ( Application of 14 C-labeled SF to animal cell lines revealed that extracellular SF was rapidly depleted in the medium and that intracellular enrichment of SF was driven by conjugation of SF to GSH

Glycine inhibits ethanol-induced oxidative stress, neuroinflammation and apoptotic neurodegeneration in postnatal rat brain

& Skerra, A

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