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10.1055/s-1992-26123

1,4 Hyperglycemia, dyslipidemia, and insulin resistance activate various metabolic pathways, resulting in oxidative stress, mitochondrial dysfunction, inflammation, and microvascular damage, culminating in neuronal injury, Schwann cell damage, and myelin sheath degeneration

These findings highlight the potential of combination strategies to maximize metabolic benefits, warranting further investigation in clinical studies

Dietary supplementation of L -carnitine ameliorates metabolic syndrome independent of trimethylamine N -oxide produced by gut microbes in high-fat diet-induced obese mice