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Overall, this review should be viewed as hypothesis-generating, mapping candidate pathways and highlighting priorities for well-designed intervention studies to test these mechanisms and strengthen causal inference
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Introduction Atopic diseases such as eczema, food allergy, allergic rhinitis, asthma, and eosinophilic esophagitis (EoE) share environmental and genomic risk factors, aero- and food allergen triggers, and immunological mediators and responses
