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In addition, HIF-1 can regulate numerous signaling pathways, such as GSK3/mitochondrial PTP, -catenin, ERK1/2, Bcl-2, PI3K/AKT and mTOR, which are involved in the regulation of broad-spectrum cell functions, and thus can decrease the severity of MIRI (5457)

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The authors conclude that the indicated SOD2 and SOD3 polymorphisms may be genetic risk factors for the susceptibility and progression of vitiligo suggesting the increased activity of SOD isoforms caused by the variant gene polymorphisms may lead to the accumulation of H 2 O 2 in cytoplasmic, mitochondrial and extracellular compartments resulting in melanocyte oxidative damage