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[30] Serious side effects are rare

We surmise that, on the one hand, KIRC cells may obtain more acetyl-CoA for lipid synthesis because NNMT upregulation can restrain polyamine catabolism by downregulating SAM levels, where MAT1A (S-adenosylmethionine synthase isoform type-1) and MAT2A ( S -adenosylmethionine synthase isoform type-2) expression levels are similar to normal kidney cells [57]

Comparative Studies Laboratories use this fragment as a benchmark to compare the efficacy of various growth hormone analogs and other metabolic regulators in preclinical studies

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