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Description
In agreement, glycogen synthase kinase-3, which demonstrates increased kinase activity in AD, is able to directly phosphorylate Nrf2, thereby inducing its nuclear efflux
Regulation and metabolic functions of mTORC1 and mTORC2
When systemic mitochondrial dysfunction occurs, the neurological consequences are severe
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Inhibits the accumulation of ammonia, which promotes endurance capacity and reduction of muscle damage
