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Developed steatosis further stimulates de novo lipogenesis, a characteristic feature of MASLD patients, while concurrently reducing lipid efflux in the form of high-density lipoprotein (HDL) particles due to insufficient apoB protein synthesis [33]

C32E8.9 is required for the immune response activated by mitochondrial translation inhibition To untangle how C32E8.9 plays a role in mitochondrial translation-mediated lifespan extension without significantly altering UPR mt , we performed RNA sequencing on total RNA isolated from day 5 adult worms fed with mrps-5 RNAi, C32E8.9 RNAi, or the combination (Fig

This enhanced uptake underscores the potential of surface modification of PLGA nanoparticles using chitosan to improve cellular interaction and CNS targeting via the intranasal route