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[DOI] [PMC free article] [PubMed] [Google Scholar] 45.Ortel TL, Neumann I, Ageno W, et al
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In BM-MSCs, METTL14 regulated osteogenic differentiation via the mA/IGF2BPs/Beclin-1 signaling axis [126], while METTL16 suppressed osteogenesis by promoting mA modification of PPAR transcripts, thereby enhancing its expression [127]
24 There are many hallmarks of cell aging that could influence ECM synthesis and breakdown and thus contribute to the development of tendinopathy
