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doi: 10.1007/BF00427441
In vitro studies demonstrate that cancer-associated fibroblasts (CAFs) support hormone-sensitive prostate cancer (HSPC) proliferation via glutamine and -KG secretion, fueling energy and biosynthetic demands (194, 195)
Current evidence indicates that empagliflozin demonstrates a promising ability to protect against bleomycin-induced PF in rats by inhibiting ferroptosis via modulating the Sesn2/AMPK/Nrf2/HO-1 signaling pathway
Yes, and that chemical identity is the key to both its power and its fragility
