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Widespread use of targeted therapies in the clinic requires high affinity, tumor-specific agents as effective targeting vehicles to deliver therapeutics to the tumor sites
In mice, Bmal1 promotes the expression of antioxidant enzymes and plays an autonomous role in maintaining neural integrity, such that its reduction induces oxidative stress, neuroinflammation, and synaptic dysfunction independently of behavioral rhythmic changes (Griffin et al., 2020
10.4081/ejtm.2025.13690 126 EmeryD
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