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Numerous previous investigations have suggested that increased pro-inflammatory cytokine levels are crucial in the development of MTX-induced intestinal damage (Tunal-Akbay et al
Journal of Experimental Pharmacology, 4, 149-155

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BT was found to modulate individual LCL mitochondrial respiration to a common set-point, with this set-point slightly higher for the AD-A LCLs as compared to the other groups
