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While more human clinical trials are needed, anecdotal reports and real-world results are encouraging

Redick SD, Settles DL, Briscoe G, Erickson HP

This mechanistic link explains why oxidative stress can amplify SASP-mediated tissue damage, creating a self-reinforcing cycle of inflammation and cellular dysfunction.18 Senolytics reduce SASP not only by eliminating the senescent cells that produce high ROS but also by disrupting pro-survival signaling pathways that sustain SASP activity

It is worth emphasising that elevated B12 levels are more commonly associated with other conditions unrelated to fatty liver, including myeloproliferative disorders, certain cancers, renal failure, and excessive B12 supplementation