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Description
The data on rhIGF-1 support the mechanistic rationale that an IGF-1 analog like LR3 (which has extended half-life and greater receptor availability in research settings) could enhance protein synthesis, stimulate cellular proliferation pathways and promote more efficient nutrient partitioning, though direct clinical trials of IGF-1 LR3 itself in humans are lacking
While no supplement can completely prevent this, supporting collagen synthesis through adequate glycine availability gives your skin the building blocks it needs to remodel as your body composition changes

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Biomolecules 2021, 11, 1276
