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The researchers proposed that the senolytic effect observed in these cells is attributable not only to the disruption of the PI3K/AKT pathway but also to interference with the ERK/MEP pathway, which plays a crucial role in regulating inflammation and resistance to apoptosis [123]
Studies demonstrate BPC-157 treatment restores epithelial thickness toward normal values, objectively documenting tissue regeneration rather than merely symptomatic improvement
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