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However, GSK-3/, a central regulator in lung SCC cells, can promote the expression of GLS and glutamine metabolism by upregulating c-MYC and c-JUN to make cancer cells adapt to chronic mTOR and glycolysis inhibition (85)

This means increased protein synthesis thus promoting growth and fat loss simultaneously

Involvement of mitochondria in acetaminophen-induced apoptosis and hepatic injury: roles of cytochrome c, Bax, Bid and caspases
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Role of fatty acid uptake and fatty acid beta-oxidation in mediating insulin resistance in heart and skeletal muscle
